Background: During high-intensity and short duration exercise (≥10 seconds) phosphocreatine is primarily responsible for re-synthesize ATP, but the amount of phosphocreatine stored in
the muscle can be a limiting factor during high-intensity exercise in humans, 90% of creatine is stored in the skeletal muscle, and 30% to 40% is in its phosphorylated form. Furthermore
phosphocreatine is in greater need to quick strength, the creatine supplementation is donating a phosphate group, it is catalysed by the enzyme creatine kinase. It is believed that creatine
supplementation may have ergogenic effect on the physical performance, especially in anaerobic exercises with intermittent character. So, the aim of this study was to identify whether creatine
supplementation leads to changes in an anaerobic power.
Methods: There were 20 practitioners with the strength training 4.2 ± 1.3 of years, all males 23 ± 3.2 years old, 1.72±0.12 m, 73 ± 3.5 kg, 12 ± 2.1% body fat, no athlete reported
using creatine or some other ergogenic aid in a 3 month window. The subjects were submitted to two different protocols. The protocol 1 (P1) consisted in using placebo, protocol 2 (P2) consisted in
using 300 mg / kg / day of creatine. Both protocols supplementation occurred for 7 days. On the seventh day it was held the anaerobic evaluation protocol, which were collected Peak Power data (PP),
Average Power (AP), Fatigue Index (FI) using a cycle ergometer of the lower limbs. It calculated the % performance loss by subtracting the PP between shots. The protocol began with a heat 5 minutes
without overload cadence of 30 km/h. At the end of the heating protocol was initiated, consisting of 5 shots speed with the duration of 8 seconds and 1 minute interval between shots. The protocol
consisted in increasing overload by 8%, 9%, 10%, 11% and 12% of body weight of the subject, the protocol ended with the subjects cycling for five minutes to return to calm. It was performed ANOVA,
post hoc Bonferroni (p<0.05) to determine if there were differences in the anaerobic power between the two different supplementation protocols.
Results: It was observed that PP (F(1,38)=143.2; p=0.018) in P2 is higher than P1 especially for stage 3 (p=0.039) and 5 (p=0.02), stage 3 PP was P1=7.1±0.5 W.kg-1 and P2=7.7±1 W.kg-1
and in stage 5 was the PP: P2=7.0±0.8 W.kg-1 and P1=6.5±0.5 W.kg-1. It was found higher values of AP (F(1,38)=123.2; p=0.001) P2 to P1 when compared to stage 3 (p=0.03), the AP values were P2=4.2±0.9
W.kg-1 and P1=3.7±0.5 W.kg-1. There was no statistical difference for FI and between stages for the protocols evaluated individually. By identifying the % performance loss, it was observed that there
was a greater decrease in performance especially for PP: P1 (-33±21.6%) than P2 (-50.1±15.2%) ( p=0.018).
Conclusions: The creatine supplementation presents greater responses in peak power and average power than the placebo group, especially from the third speed shooting. Creatine
supplementation is associated with the maintenance of anaerobic power response over progressive test.